Navegando por Autor "Yue, Xu"

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    Artigo Científico
    All-cause, cardiovascular, and respiratory mortality and wildfire-related ozone: a multicountry two-stage time series analysis
    (2024) Chen, Gongbo; Guo, Yuming; Yue, Xu; Xu, Rongbin; Yu, Wenhua; Ye, Tingting; Tong, Shilu; Gasparrini, Antonio; Bell, Michelle L; Armstrong, Ben; Schwartz, Joel; Jaakkola , Jouni J K; Lavigne , Eric; Saldiva, Paulo Hilario Nascimento; Kan, Haidong; Royé, Dominic; Urban, Aleš; Vicedo-Cabrera, Ana Maria; Tobias, Aurelio; Forsberg, Bertil; Sera, Francesco; Lei, Yadong; Abramson, Michael J; Li, Shanshan
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    Artigo Científico
    Wildfire-related PM2.5 and cardiovascular mortality: A difference-in-differences analysis in Brazil
    (2024) Gao, Yuan; Huang, Wenzhong; Yu, Pei; Xu, Rongbin; Gasevic, Danijela; Yue, Xu; Coêlho, Micheline de Sousa Zanotti Stagliorio; Saldiva, Paulo Hilario Nascimento; Guo, Yuming; Li, Shanshan
    Brazil has experienced unprecedented wildfires recently. We aimed to investigate the association of wildfire-related fine particulate matter (PM2.5) with cause-specific cardiovascular mortality, and to estimate the attributable mortality burden. Exposure to wildfire-related PM2.5 was defined as exposure to annual mean wildfire-related PM2.5 concentrations in the 1-year prior to death. The variant difference-in-differences method was employed to explore the wildfire-related PM2.5-cardiovascular mortality association. We found that, in Brazil, compared with the population in the first quartile (Q1: ≤1.82 μg/m3) of wildfire-related PM2.5 exposure, those in the fourth quartile (Q4: 4.22–17.12 μg/m3) of wildfire-related PM2.5 exposure had a 2.2% (RR: 1.022, 95% CI: 1.013–1.032) higher risk for total cardiovascular mortality, 3.1% (RR: 1.031, 95% CI: 1.014–1.048) for ischaemic heart disease mortality, and 2.0% (RR: 1.020, 95% CI: 1.002–1.038) for stroke mortality. From 2010 to 2018, an estimation of 35,847 (95% CI: 22,424–49,177) cardiovascular deaths, representing 17.77 (95% CI: 11.12–24.38) per 100,000 population, were attributable to wildfire-related PM2.5 exposure. Targeted health promotion strategies should be developed for local governments to protect the public from the risk of wildfire-related cardiovascular premature deaths.
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    Artigo Científico
    Wildfire-sourced fine particulate matter and preterm birth risks in Brazil: A nationwide population-based cohort study
    (2024) Zhang, Yiwen; Huang, Wenzhong; Xu, Rongbin; Ye, Tingting; Chen, Gongbo; Yue, Xu; Coêl , Micheline de Sousa Zanotti Stagliorio; Saldiva, Paulo Hilario Nascimento; Song, Jiangning; Guo, Yuming; Li, Shanshan
    Wildfire-specific particulate matter with diameters ≤ 2.5 µm (PM2.5) is the key component of wildfire smoke, with potentially higher toxicity than PM2.5 from other sources. In this nationwide population-based cohort study, we included 22,163,195 births from Brazil during 2010–2019. Daily wildfire-specific PM2.5 was estimated through the chemical transport model. Time-varying Cox proportional hazards models were used to characterize the exposure-time-response (E-T-R) relationship between weekly wildfire-specific PM2.5 exposure and preterm birth (PTB) risks, followed by subgroup analyses. A 10 µg/m3 increment in wildfire-specific PM2.5 was associated with a hazard ratio of 1.047 (95 % confidence interval [CI]: 1.032–1.063) for PTB. Stronger associations between wildfire-specific PM2.5 and PTB were observed during earlier pregnancy, among female infants, and pregnant women < 18 years old, in ethnic minorities, with a length of education ≥ 11 years, from low-income or high temperature municipalities, and residing in North/Northeast regions. An estimated 1.47 % (95 % CI: 1.01 %–1.94 %) of PTBs were attributable to wildfire-specific PM2.5 in Brazil, increasing from 2010 to 2019. The PTBs attributable to wildfire-specific PM2.5 surpassed those attributed to non-wildfire PM2.5 (0.31 %, 95% CI: 0.09 %–0.57 %). Wildfire emerged as a critical source contributing to the PM2.5-linked PTBs. Prioritized fire management and emission control strategies are warranted for PTB prevention.